Showing items 1 - 19 of 19

Your selections:

Alveolar epithelial cells of lung fibrosis patients are susceptible to severe virus-induced injury

A Persistent Neutrophil-Associated Iimmune Signature Characterizes Post-COVID-19 Pulmonary Sequelae

Substrate stiffness engineered to replicate disease conditions influence senescence and fibrotic responses in primary lung fibroblasts

A cGAS-dependent response links DNA damage and senescence in alveolar epithelial cells: a potential drug target in IPF

A senescence bystander effect in human lung fibroblasts

Ageing mechanisms that contribute to tissue remodeling in lung disease

Regulation of cellular senescence is independent from profibrotic fibroblast-deposited ECM

Epithelial mesenchymal transition in respiratory disease: fact or fiction

Regulation of cellular senescence by extracellular matrix during chronic fibrotic diseases

Self DNA perpetuates IPF lung fibroblast senescence in a cGAS-dependent manner

Senescence of IPF lung fibroblasts disrupt alveolar epithelial cell proliferation and promote migration in wound healing

Modeling the impact of low-dose particulate matter on lung health

STAT3 regulates the onset of oxidant-induced senescence in lung fibroblasts

Fibroblast senescence in the pathology of idiopathic pulmonary fibrosis

Mitochondrial dysfunction contributes to the senescent phenotype of IPF lung fibroblasts

The fibrogenic actions of the coagulant and plasminogen activation systems in pulmonary fibrosis

Annexin A2 contributes to lung injury and fibrosis by augmenting factor Xa fibrogenic activity

The fibrogenic actions of lung fibroblast-derived urokinase: a potential drug target in IPF

The coagulant factor Xa induces protease-activated receptor-1 and annexin A2-dependent airway smooth muscle cytokine production and cell proliferation

  • 1
  • Disclaimer
  • Privacy
  • Copyright
  • Contact
  • Deposit